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A HORSE IS PRESENTED TO YOUR CLINIC WITH A HISTORY OF SHIFTING LAMENESS, FIRM BONEY PROMINENCES AROUND THE FRONTONASAL SUTURES AND PAIN WHEN EATING.

A HORSE IS PRESENTED TO YOUR CLINIC WITH A HISTORY OF SHIFTING LAMENESS, FIRM BONEY PROMINENCES AROUND THE FRONTONASAL SUTURES AND PAIN WHEN EATING.

A horse is presented to your clinic with a history of shifting lameness, firm boney
prominences around the frontonasal sutures and pain when eating. The horse is
suspected of having nutritional secondary hyperparathyroidism (NSH).
Answer all parts of this question:
a) Describe in detail the pathophysiology of NSH. You may use diagrams.
(12.5 marks)
b) Describe in detail the diagnosis of NSH. (12.5 marks)
c) Describe the management of the case described above. (5 marks)
6. Answer both parts of this question:
a) List the clinical signs associated with hepatic insufficiency and failure in the
horse, and for each clinical sign, describe in detail the pathophysiologic basis of
these manifestations. (20 marks)
b) Discuss in detail the management of hepatic encephalopathy. (10 marks)
Continued over page
Equine Medicine Paper 2 Page 7 of 8
7. A 450 kg two-year-old Thoroughbred colt was presented with acute onset severe
watery diarrhoea. Pertinent findings from a thorough physical examination revealed
moderate signs of depression, significant tachycardia between 80–100 bpm, pyrexia of
39.1°C, and dark injected mucous membranes with CRT of around 3 seconds. Small
petechial haemorrhages were also noted on the lower lip mucous membranes. The
colt’s peripheral pulses were mildly weak and his lower limbs felt cool to the touch.
His digital pulses were not elevated at the time of presentation. The colt urinated a
small volume at presentation and the urine was thick with a dark red tinge.
Stall side laboratory analysis performed on a venous sample at presentation yielded
the abnormal results as below:


 

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